Blue light exposure causes retinal to trigger reactions that generate poisonous chemical molecules in photoreceptor cells. If you shine blue light on retinal, the retinal kills photoreceptor cells as the signalling molecule on the membrane dissolves," said Kasun Ratnayake, a PhD student researcher working in KarunarathneChina wholesale led vapor tight fixture group."Photoreceptor cells do not regenerate in the eye.The study, published in the journal Scientific Reports, found that blue light exposure causes retinal to trigger reactions that generate poisonous chemical molecules in photoreceptor cells."It's toxic.end-ofTags: smartphones, digital devices, blindness, retinaLocation: United States, Washington."No activity is sparked with green, yellow or red light.

Macular degeneration, an incurable eye disease that results in significant vision loss starting on average in a person's 50s or 60s, is the death of photoreceptor cells in the retina. When they're dead, they're dead for good," said Ratnayak.Washington: Blue light emitted from smartphones and other digital devices can accelerate blindness by transforming vital molecules in the eye's retina into cell killers, a study has found."We are being exposed to blue light continuously, and the eye's cornea and lens cannot block or reflect it," said Ajith Karunarathne, an assistant professor at University of Toledo in the US. Blue light alone or retinal without blue light had no effect on cells.Blue light emitted from smartphones and other digital devices can accelerate blindness by transforming vital molecules in the eye's retina into cell killers, a study has found.When exposed to blue light, these cell types died as # a result of the combination with retinal."If you look at the amount of light coming out of your cell phone, it's not great but it seems tolerable," Dr.

The retinal-generated toxicity by blue light is universal."It's no secret that blue light harms our vision by damaging the eye's retina. However, as a person ages or the immune system is suppressed, people lose the ability to fight against the attack by retinal and blue light. Our experiments explain how this happens, and we hope this leads to therapies that slow macular degeneration, such as a new kind of eye drop," said Karunarathne. The researcher found that a molecule called alpha tocoferol, a Vitamin E derivative and a natural antioxidant in the eye and body, stops the cells from dying. Those cells need molecules called retinal to sense light and trigger a cascade of signalling to the brain."Some cell phone companies are adding blue-light filters to the screens, and I think that is a good idea," said John Payton, visiting assistant professor at University of Toledo. Karunarathne introduced retinal molecules to other cell types in the body, such as cancer cells, heart cells and neurons

In the future, it is planned to disseminate this light also within a European network. With a linewidth of only 10 mHz, the laser that researchers from the Physikalisch-Technische Bundesanstalt (PTB) have now developed together with US researchers from JILA, a joint institute of the National Institute of Standards and Technology (NIST) and the University of Colorado, Boulder, has established a new world record. For researchers, this is a measure for the light wave&China led Tri proof lights39;s regular frequency and linewidth. The thermal noise of the silicon body is so low that the length fluctuations observed only originate from the thermal noise of the dielectric SiO2/Ta2O5 mirror layers. Within the scope of a nearly 10-year-long joint project with the US colleagues from JILA in Boulder, Colorado, a laser has now been developed at PTB whose linewidth is only 10 mHz (0. With novel crystalline mirror layers and lower temperatures, the disturbing thermal noise can be further reduced. The results have been published in the current issue of "Physical Review Letters".

"The smaller the linewidth of the laser, the more accurate the measurement of the atom's frequency in an optical clock. Lasers have brought about a real revolution in many fields of research and in metrology – or even made some new fields possible in the first place. More than 50 years have passed since the first technical realization of the laser, and we cannot imagine how we could live without them today. Similar to an organ pipe, the resonator length determines the frequency of the wave which begins to oscillate, i.Lasers were once deemed a solution without problems – but that is now history. At PTB, the ultrastable light from these lasers is already being distributed via optical waveguides and is then used by the optical clocks in Braunschweig. This length corresponds to nearly ten times the distance between the Earth and the moon. Although the mirror layers are only a few micrometers thick, they dominate the resonator's length stability., the light wave inside the resonator. Its extent depends on the materials used to build the resonator as well as on the resonator's temperature.

This precision is useful for various applications such as optical atomic clocks, precision spectroscopy, radioastronomy and for testing the theory of relativity. In total, the resonator length, however, only fluctuates in the range of 10 attometers.01 Hz), hereby establishing a new world record. 3. The linewidth could then even become smaller than 1 mHz.In addition to the new laser’s extremely small linewidth, Legero and his colleagues found out by means of measurements that the emitted laser light's frequency was more precise than what had ever been achieved before.The scientists at PTB had to isolate the resonator nearly perfectly from all environmental influences which might change its length.The core piece of each of the lasers is a 21-cm long Fabry-Pérot silicon resonator. The resonator consists # of two highly reflecting mirrors which are located opposite each other and are kept at a fixed distance by means of a double cone. This plan would allow even more precise comparisons between the optical clocks in Braunschweig and the clocks of our European colleagues in Paris and London", Legero says.

end-ofTags: smartphones.When these cells are exposed to ongoing light, a protein called melanopsin continually regenerates within them, signalling levels of ambient light directly to the brain to regulate consciousness, sleep and alertness. "Our study suggests the two arrestins accomplish regeneration of melanopsin in a peculiar way," Panda said. The results, published in the journal Cell Reports, may lead to new treatments for migraines, insomnia, jet lag and circadian rhythm disorders.They discovered that some of these cells have the ability to sustain light responses when exposed to repeated long pulses of light, while others become desensitised. Melanopsin plays a pivotal role in synchronising our internal clock after 10 minutes of illumination and, under bright light, suppresses the hormone melatonin, responsible for regulating sleep.The reason, it turns out, is that arrestin helps melanopsin regenerate in the retinal cells."Compared to other light-sensing cells in the eye, melanopsin cells respond as long as the light lasts, or even a few seconds longer," said staff scientist Ludovic Mure."

This lifestyle causes disruptions to our circadian rhythms and has deleterious consequences on health," Panda said. Conventional wisdom has held that proteins called arrestins, which stop the activity of certain receptors, should halt cells&ip65 industrial lighting 39; photosensitive response within seconds of lights coming on.The backs of our eyes contain a sensory membrane called the retina, whose innermost layer contains a tiny subpopulation of light-sensitive cells that operate like pixels in a digital camera."When these two steps are done in quick succession, the cell appears to respond continuously to light," he said. In mice lacking either version of the arrestin # protein (beta-arrestin 1 and beta-arrestin 2), the melanopsin-producing retinal cells failed to sustain their sensitivity to light under prolonged illumination. The researchers used molecular tools to turn on a production of melanopsin in retinal cells in mice."One arrestin does its conventional job of arresting the response, and the other helps the melanopsin protein reload its retinal light-sensing co-factor.Researchers found that cells in the eye process ambient light and reset our internal clocks, the daily cycles of physiological processes.

When these cells are exposed to artificial light late into the night, our internal clocks can get confused, resulting in a host of health issues.The researchers were surprised to find that arrestins are in fact necessary for melanopsin to continue responding to prolonged illumination. "We are continuously exposed to artificial light, whether from screen time, spending the day indoors or staying awake late at night," said Professor Satchin Panda from Salk Institute.(Representational image)Scientists have uncovered how artificial light from smartphones and computers can disrupt sleep, a finding which may lead to new treatments for migraines, insomnia, jet lag and circadian rhythm disorders.These disorders have been tied to cognitive dysfunction, cancer, obesity, insulin resistance, metabolic syndrome and more, researchers said."That's critical because our circadian clocks are designed to respond only to prolonged illumination," Mure said.Researchers at Salk Institute in the US found that certain cells in the eye process ambient light and reset our internal clocks, the daily cycles of physiological processes known as the circadian rhythm.